Includes bibliographical references and indexes.
|Statement||edited by Ronald Glaser, Tamar Gotlieb-Stematsky.|
|Series||Infectious diseases and antimicrobial agents ;, 2|
|Contributions||Glaser, Ronald, 1939-, Gotlieb-Stematsky, Tamar.|
|LC Classifications||RC147.H6 H85|
|The Physical Object|
|Pagination||ix, 280 p. :|
|Number of Pages||280|
|LC Control Number||81019423|
One of the most important features of the book is that it covers aspects of both basic research and clinical medicine. Herpesviridae, a family of double-strand DNA viruses, has unique biological features by which these viruses establish latency after primary infection and reactivate in later life. Exanthem Subitum and Human Herpesvirus 6 (HHV-6) Infection. Koichi Yamanishi, Toshio Kondo, Kazukiro Kondo, Yasuhiko Hayakawa, Shinichi Kido, Kazuo Takahashi et al. Pages Viral Pathology of Human Herpesvirus 6 Infection. ISBN: OCLC Number: Description: ix, pages: illustrations ; 24 cm. Contents: Herpes simplex virus infections / Ervin Adam --Human cytomegalovirus infections / John L. Sullivan and James B. Hanshaw --Varicella-zoster virus infections / Charles Grose --Epstein-Barr virus and infectious monoucleosis / Werner Henle and Gertrude Henle --Association of Epstein. Red Book: ; Red Book: Errata; Red Book: ; Red Book: Errata; Red Book: ; Infection Control and Prevention for Hospitalized Children Human Herpesvirus 8 p Human Immunodeficiency Virus Infection p I.
Human herpesviruses type 6 (HHV-6) and type 7 (HHV-7) are ubiquitous, lymphotropic, β-herpesviruses. 1, 2 They are 2 of the known causative agents of roseola infantum, also known as exanthem subitum. 3, 4 It is now understood that in otherwise healthy children, the spectrum of diseases associated with HHV-6 and HHV-7 is far broader than the benign illness of roseola. Epstein–Barr virus infection after adolescence and age independent human herpesvirus 6A infection are risk factors for multiple sclerosis. Clinical manifestations of primary infection with human herpesvirus 6B (HHV-6B) include roseola (exanthem subitum) in approximately 20% of infected children, as well as a nonspecific febrile illness without rash or localizing signs. Acute HHV-6B infection may be accompanied by cervical and characteristic postoccipital lymphadenopathy, gastrointestinal tract or respiratory tract signs, and. Overview of Herpesvirus Infections By. Kenneth M. Kaye, MD, Brigham and Women’s Hospital, Harvard Medical School (EBV) and human herpesvirus type 8 (HHV-8), also known as Kaposi sarcoma–associated herpesvirus (KSHV), can cause certain cancers. Roseola infantum is a childhood disease caused by herpesvirus 6 (and sometimes 7).
Herpesvirus group can establish primary infections with nonsevere symptoms, which can result in an efficient immune response that prevents a new infection. However, the virus is not completely eliminated, its genome remains within cells without productive : Ronaldo Luis Thomasini. Thomas M. Donnelly BVSc, Diplomate ACLAM, in Ferrets, Rabbits, and Rodents (Second Edition), Viral Infections. Chinchillas are susceptible to human herpesvirus 1 and may play a role as a temporary reservoir for human infections. Goudas and Giltoy 22 described a spontaneous, herpeslike viral infection in a female chinchilla. Wohlsein et al. 95 reported a 1-year-old male chinchilla with a. There is no loftier aim than to cure or prevent human infections with these viruses. The objective of much of the current research on herpesviruses is directed toward an understanding of the molecular mechanisms involved in initiation of infection, establish ment and termination of latent state, virus multiplication, and the destruction of. The spectrum of viruses known to cause encephalitis is continuously growing. Lately, human herpesvirus 6 variants A and B (HHV-6A and HHV-6B) (Salahuddin et al. ) and HHV-7 (Frenkel et al. ) (Herpesviridae family, Betaherpesvirinae subfamily, Roseolovirus genus) have been identied.